Matt Quinn, PhD
- Assistant Professor, Pathology - Comparative Medicine

Matt Quinn, PhD
Research Interests
- Obesity
- Women's Health
- Fatty Liver
- Nuclear Receptors
- Reproduction
- Epigenesis, Genetic
- Education
- BA, Texas Tech University, 2008
- PhD, Texas A&M University, 2013
- Fellowship
- Molecular Endocrinology, National Institute of Environm, 2018
- Memberships
- Endocrine Society
- Positions
- Assistant Professor, Pathology - Comparative Medicine
- Assistant Professor, Molecular Medicine
- Departments and Affiliations
- Pathology - Comparative Medicine
- Molecular Medicine
- Center on Diabetes, Obesity, and Metabolism
- Center for Precision Medicine
- Center for Redox Biology and Medicine
- Sticht Center for Healthy Aging and Alzheimer’s Prevention
- Cardiovascular Sciences Center
Research
- Silencing of maternal hepatic glucocorticoid receptor is essential for normal fetal development in mice. Quinn MA, McCalla A, He B, Xu X, Cidlowski JA. Commun Biol. 2019; 2:104.
- Comms Bio. In Press. Quinn MA, McCalla A, He B, Xu X, Cidlowski JA. Silencing of maternal hepatic glucocorticoid receptor is essential for normal fetal development in mice. 2019 Jan; 1;
- Estrogen Deficiency Promotes Hepatic Steatosis via a Glucocorticoid Receptor-Dependent Mechanism in Mice. Quinn MA, Xu X, Ronfani M, Cidlowski JA. Cell Rep. 2018 03; 22(10):2690-2701.
- Bile Acid Signaling Is Involved in the Neurological Decline in a Murine Model of Acute Liver Failure. McMillin M, Frampton G, Quinn M, Ashfaq S, de los Santos M, Grant S, DeMorrow S. Am. J. Pathol. 2016 Feb; 186(2):312-23.
- Endogenous hepatic glucocorticoid receptor signaling coordinates sex-biased inflammatory gene expression. Quinn MA, Cidlowski JA. FASEB J. 2016 Feb; 30(2):971-82.